More specifically, the team analyzed how low levels of PRDM2 impact the way memories are processed and stored– a process known as “consolidation.” And they found that down-regulation of PRDM2 actually increases the storage of fearful memories.
Unfortunately, though, researchers do not currently have a way of increasing PRDM2 to weaken or erase fear memories. Nonetheless, Barbier still believes this finding is groundbreaking in further illuminating chronic fear response processes.
“The mechanism may be part of the explanation of why some individuals have a greater vulnerability to developing anxiety-related conditions. It may also explain why these conditions and alcohol dependence so often are present together,” she said.
Still, further research regarding if and how PRDM2 levels might be manipulated to lessen fear responses is warranted.
To read the study’s complete findings, which have since been published in Molecular Psychiatry, visit the link here.
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