“This study helped unravel the puzzle of junk DNA– showing how these repetitive sequences can block DNA replication and repair,” explained Kristian Helin, the Chief Executive of ICR.
“It’s possible that this mechanism could play a role in the development of cancer as a cause of genetic instability– especially as cancer cells start dividing more quickly and place the process of DNA replication under more stress.”
Now equipped with these DNA replication findings, the researchers hope their improved understanding will lead to new cancer treatment options.
“Understanding the mechanisms underlying genetic mutation and instability is critical if we are to find innovative new ways to treat cancer that exploit fundamental weaknesses in cancer cells,” Helin added.
To read the study’s complete findings which have since been published in Nature Communications, visit the link here.
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